Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice
JP Friedmann Angeli, M Schneider, B Proneth…�- Nature cell�…, 2014 - nature.com
Ferroptosis is a non-apoptotic form of cell death induced by small molecules in specific
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice
JP Friedmann Angeli, M Schneider, B Proneth…�- Nature Cell�…, 2014 - hero.epa.gov
Ferroptosis is a non-apoptotic form of cell death induced by small molecules in specific
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice
JPF Angeli, M Schneider, B Proneth…�- Nature Cell�…, 2014 - researchers.mq.edu.au
Ferroptosis is a non-Apoptotic form of cell death induced by small molecules in specific
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice.
JP Friedmann Angeli, M Schneider…�- Nature Cell�…, 2014 - search.ebscohost.com
Ferroptosis is a non-apoptotic form of cell death induced by small molecules in specific
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice
JPF Angeli, M Schneider, B Proneth…�- Nature cell�…, 2014 - pubmed.ncbi.nlm.nih.gov
Ferroptosis is a non-apoptotic form of cell death induced by small molecules in specific
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice
JPF Angeli, M Schneider, B Proneth, YY Tyurina…�- Nature Cell�…, 2014 - go.gale.com
Ferroptosis is a non-apoptotic form of cell death induced by small molecules in specific
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice
JP Friedmann Angeli, M Schneider, B Proneth…�- Nature Cell�…, 2014 - orca.cardiff.ac.uk
Ferroptosis is a non-apoptotic form of cell death induced by small molecules in specific
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
[CITATION][C] Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice
AJP Friedmann, B Proneth, S Kobayashi, T Seibt…�- Nature Cell�…, 2014 - elibrary.ru
Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice.
FA JP, M Schneider, B Proneth, YY Tyurina…�- Nature Cell�…, 2014 - europepmc.org
Ferroptosis is a non-apoptotic form of cell death induced by small molecules in specific
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice.
JP Friedmann Angeli, M Schneider, B Proneth… - 2014 - mediatum.ub.tum.de
Ferroptosis is a non-apoptotic form of cell death induced by small molecules in specific
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…
tumour types, and in engineered cells overexpressing oncogenic RAS. Yet, its relevance in�…