About
Titles
Instructor
Appointments
Dermatology
InstructorPrimary
Other Departments & Organizations
Education & Training
- Transitional Year
- Mercy Catholic Medical Center (2020)
- MD/PhD
- Penn State College of Medicine
- BA
- Rutgers University, Biology (2009)
- AA
- Union County College, Biology (2007)
Research
Overview
Medical Subject Headings (MeSH)
Hair Follicle; Immune System
ORCID
0000-0002-3279-5974
Research at a Glance
Yale Co-Authors
Frequent collaborators of Michal Kidacki's published research.
Publications Timeline
A big-picture view of Michal Kidacki's research output by year.
Matthew Vesely, MD, PhD
Francesc Lopez-Giraldez, PhD
Jonathan Leventhal, MD
William Damsky, MD, PhD
17Publications
173Citations
Publications
2023
207 Spatial proteomic and transcriptomic profiling of lichen planus reveals a unique inflammatory architecture
Garritano J, Kidacki M, Cho C, Lopez-Giraldez F, Damsky W, Chen L, Vesely M. 207 Spatial proteomic and transcriptomic profiling of lichen planus reveals a unique inflammatory architecture. Journal Of Investigative Dermatology 2023, 143: s367. DOI: 10.1016/j.jid.2023.09.215.Peer-Reviewed Original ResearchImmune Inhibitory Molecule PD-1 Homolog (VISTA) Colocalizes with CD11b Myeloid Cells in Melanoma and Is Associated with Poor Outcomes
Vesely M, Kidacki M, Gaule P, Gupta S, Chan N, Han X, Yeung J, Chen L. Immune Inhibitory Molecule PD-1 Homolog (VISTA) Colocalizes with CD11b Myeloid Cells in Melanoma and Is Associated with Poor Outcomes. Journal Of Investigative Dermatology 2023, 144: 106-115.e4. PMID: 37562584, DOI: 10.1016/j.jid.2023.07.008.Peer-Reviewed Original ResearchAltmetricMeSH Keywords and ConceptsConceptsCD11b myeloid cellsImmune inhibitory moleculesPD-L1 expressionVISTA expressionMyeloid cellsFuture potential therapeutic targetsPD-L1/B7Tumor microenvironmentInhibitory moleculesMultiplexed quantitative immunofluorescencePrimary cutaneous melanomaImmunosuppressive tumor microenvironmentNegative prognostic biomarkerCurrent clinical trialsPotential therapeutic targetCause mortalityCritical cell typesPD-L1Poor outcomeTreatment of cancerMelanoma recurrenceCutaneous melanomaClinical trialsPrognostic biomarkerT cells
2021
Iatrogenic calcinosis cutis from extravasated phosphate-containing solution treated with topical sodium thiosulfate
Xie CB, Kidacki M, Ring N, Panse G, Leventhal JS. Iatrogenic calcinosis cutis from extravasated phosphate-containing solution treated with topical sodium thiosulfate. JAAD Case Reports 2021, 17: 31-33. PMID: 34692964, PMCID: PMC8517711, DOI: 10.1016/j.jdcr.2021.09.013.Peer-Reviewed Case Reports and Technical NotesCitationsAltmetric
2020
Twist2 is NFkB-responsive when p120-catenin is inactivated and EGFR is overexpressed in esophageal keratinocytes
Lehman H, Kidacki M, Stairs D. Twist2 is NFkB-responsive when p120-catenin is inactivated and EGFR is overexpressed in esophageal keratinocytes. Scientific Reports 2020, 10: 18829. PMID: 33139779, PMCID: PMC7608670, DOI: 10.1038/s41598-020-75866-0.Peer-Reviewed Original ResearchCitationsMeSH Keywords and ConceptsConceptsEsophageal squamous cell carcinomaCancer typesYear of diagnosisSquamous cell carcinomaEsophageal keratinocytesOverexpression of EGFRHuman esophageal keratinocytesAggressive cell typesFatal cancer typePoor prognosisCell carcinomaTwist2 expressionEGFR overexpressionEGFREMT-inducing geneNFkBResponsive targetsTwist2Paucity of researchCell typesPresent studyKeratinocytesP120-cateninOverexpressionComplete loss
2019
E‐cadherin and p120ctn protein expression are lost in hidradenitis suppurativa lesions
Nelson AM, Cong Z, Gettle SL, Longenecker AL, Kidacki M, Kirby JS, Adams DR, Stairs DB, Danby FW. E‐cadherin and p120ctn protein expression are lost in hidradenitis suppurativa lesions. Experimental Dermatology 2019, 28: 867-871. PMID: 31107992, DOI: 10.1111/exd.13973.Peer-Reviewed Original ResearchCitationsAltmetricMeSH Keywords and ConceptsConceptsProtein expressionHS lesional skinPathogenesis of HSInflammatory skin diseaseJunction proteinsHidradenitis suppurativa lesionsInflammatory skin conditionNon-lesional skinSevere skin lesionsE-cadherinAdherens junction proteinsE-cadherin expressionHurley stageAtopic dermatitisLesional skinDesmosome sizeHS pathogenesisHS skinAcne vulgarisHS patientsSkin lesionsPilosebaceous unitSkin conditionsSkin diseasesHealthy skin1028 Deciphering the mechanism of action of isotretinoin in acne patients using next gene sequencing technologies
Thiboutot D, Kidacki M, Sarfo A, Schneider A, Albert I, Longenecker A, Cong Z, Nelson A. 1028 Deciphering the mechanism of action of isotretinoin in acne patients using next gene sequencing technologies. Journal Of Investigative Dermatology 2019, 139: s178. DOI: 10.1016/j.jid.2019.03.1104.Peer-Reviewed Original Research‘Invasive proliferative gelatinous mass’ of hidradenitis suppurativa contains distinct inflammatory components
Kidacki M, Cong Z, Flamm A, Helm K, Danby FW, Nelson AM. ‘Invasive proliferative gelatinous mass’ of hidradenitis suppurativa contains distinct inflammatory components. British Journal Of Dermatology 2019, 181: 192-193. PMID: 30597519, DOI: 10.1111/bjd.17541.Peer-Reviewed Original ResearchCitationsAltmetricAn Immunohistopathologic Study to Profile the Folate Receptor Beta Macrophage and Vascular Immune Microenvironment in Giant Cell Arteritis.
Albano-Aluquin S, Malysz J, Kidacki M, Ratnam M, Olsen NJ. An Immunohistopathologic Study to Profile the Folate Receptor Beta Macrophage and Vascular Immune Microenvironment in Giant Cell Arteritis. Journal Of Visualized Experiments 2019 PMID: 30799840, DOI: 10.3791/58713.Peer-Reviewed Original ResearchCitationsAltmetricMeSH Keywords and ConceptsConceptsGiant cell arteritisFolate receptor betaCell arteritisImmune microenvironmentChronic immune-mediated diseaseImmune-mediated diseasesTemporal artery biopsyT helper lymphocytesLarge-sized arteriesPro-inflammatory responseMyeloid leukemia cellsOcclusive symptomsTotal CD68Disease activityInflammatory disease researchArtery biopsyLymphohistiocytic inflammationSynovial macrophagesSized arteriesCD3 expressionTherapeutic responseVision lossImmunohistopathologic studyPathognomonic featuresIntimal hyperplasiaAn Immunohistopathologic Study to Profile the Folate Receptor Beta Macrophage and Vascular Immune Microenvironment in Giant Cell Arteritis
Albano-Aluquin S, Malysz J, Kidacki M, Ratnam M, Olsen N. An Immunohistopathologic Study to Profile the Folate Receptor Beta Macrophage and Vascular Immune Microenvironment in Giant Cell Arteritis. Journal Of Visualized Experiments 2019 DOI: 10.3791/58713-v.Peer-Reviewed Case Reports and Technical Notes
2018
Pseudohyperphosphatemia in a patient with incidentally identified progression of smoldering myeloma
Francis ER, Chen F, Kidacki M, Miller R, Alkhasoneh M, Talamo G, Straub M, Klinefelter K, Kodali N, Zhu Y. Pseudohyperphosphatemia in a patient with incidentally identified progression of smoldering myeloma. Clinica Chimica Acta 2018, 487: 306-308. PMID: 30315756, DOI: 10.1016/j.cca.2018.10.016.Peer-Reviewed Original ResearchCitationsMeSH Keywords and ConceptsConceptsSerum phosphorus concentrationHigher serum phosphorus concentrationsKappa/lambda ratioMultiple myelomaLambda chain ratioRare laboratory findingSymptomatic multiple myelomaAppropriate laboratory investigationsLambda ratioLaboratory findingsDisease progressionOrtho VITROSSerial dilutionsCorrect diagnosisPatientsMyelomaPseudohyperphosphatemiaProgressionChain ratioCliniciansLaboratory investigationsGammaglobulinMisdiagnosisVITROSDiagnosis
Academic Achievements and Community Involvement
honor Transitional Year Excellence Award
Other AwardMercy Catholic Medical CenterDetails06/27/2020United Stateshonor Travel Grant
Other AwardSymposium on Hidradenitis Suppurativa AdvancesDetails10/09/2018United Stateshonor Travel Award
Other AwardPenn State MD/PhD ProgramDetails10/09/2018United Stateshonor Daniel Notterman Award
Other AwardPenn State UniversityDetails05/01/2016United Stateshonor Travel Award
Other AwardPenn State MD/PhD ProgramDetails04/06/2016United States