RNA modifications are integral to the regulation of RNA metabolism. One abundant mRNA
modification is N 6-methyladenosine (m6A), which affects various aspects of RNA�…

Fragile X syndrome, the most common form of inherited mental retardation, is caused by the
absence of the fragile X mental retardation protein FMRP. The RNA-binding FMRP�…

ABSTRACT RNA modifications are an emerging layer of posttranscriptional gene regulation
in eukaryotes. N6-methyladenosine (m6A) is among the most abundant modifications in�…

Fragile X mental retardation protein (FMRP) and its autosomal paralog FXR2P are selective
neuronal RNA-binding proteins, and mice that lack either protein exhibit cognitive deficits�…

Messenger RNA m6A modification is shown to regulate local translation in axons. However,
how the m6A codes in axonal mRNAs are read and decoded by the m6A reader proteins is�…

Abstract Fragile X Syndrome (FXS) is the most common form of inherited intellectual
disability and results from a loss of Fragile X mental retardation protein (FMRP). FMRP is�…

N6-methyladenosine (m6A), the most abundant modification in messenger RNAs (mRNAs),
is deposited by methyltransferases (“writers”) Mettl3 and Mettl14 and erased by�…

N 6-Methyladenosine (m 6 A) is emerging as a chemical mark that broadly affects the flow of
genetic information in various biological processes in eukaryotes. Recently, Xiao et al�…

The Drosophila polyadenosine RNA binding protein Nab2, which is orthologous to a human
protein lost in a form of inherited intellectual disability, controls adult locomotion, axon�…

During the maternal-to-zygotic transition (MZT), multiple mechanisms precisely control
massive decay of maternal mRNAs. N 6-methyladenosine (m 6 A) is known to regulate�…